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Building on this idea, we further pushed the boundaries by combining an N-terminal Rex domain from NAPstar4, a NAPstar variant initially dismissed due to lack of detectable affinity for NADPH ( Figure 1C ), with a C-terminal Rex domain from NAPstar3

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muris have reduced enzyme activity of and cell percentage staining for retinal dehydrogenase in lamina propria-derived dendritic cells and macrophages that did not rebound until the infection was cleared, indicating that chronicity may be related to decreased local RA levels (31) and impaired immune responses