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A study involving mice also found that the release of free copper ions induced neointimal thickening and contributed to the development of atherosclerotic lesions in damaged rat carotid arteries (62)

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Protein kinase C phosphorylates Ser40 of Nrf2, resulting in the activation of Nrf2 ( Figure 4B ) inhibits its activity by promoting β-TrCP-mediated ubiquitination and degradation of Nrf2 (Rada et al., 2012)